Atıf İçin Kopyala
Kurt A., TUMKAYA L., Kalkan Y., TÜRÜT H., CURE M. C., CURE E., ...Daha Fazla
IRANIAN JOURNAL OF BASIC MEDICAL SCIENCES, cilt.18, sa.11, ss.1093-1099, 2015 (SCI-Expanded)
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Yayın Türü:
Makale / Tam Makale
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Cilt numarası:
18
Sayı:
11
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Basım Tarihi:
2015
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Dergi Adı:
IRANIAN JOURNAL OF BASIC MEDICAL SCIENCES
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Derginin Tarandığı İndeksler:
Science Citation Index Expanded (SCI-EXPANDED), Scopus
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Sayfa Sayıları:
ss.1093-1099
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Anahtar Kelimeler:
Adalimumab, Endothelin-1, Ischemia reperfusion, Lung injury, Tumor necrosis factor-alpha, RHEUMATOID-ARTHRITIS, ENDOTHELIN-1, MALONDIALDEHYDE, INFLIXIMAB, APOPTOSIS, SAFETY
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Recep Tayyip Erdoğan Üniversitesi Adresli:
Evet
Özet
Objective(s): Increasing cytokines and reactive oxygen species (ROS) during ischemia reperfusion (I-R) leads to the lung damage. Adalimumab (Ada) is a potent tumor necrosis factor-alpha (TNF-alpha) inhibitor agent. We aimed to evaluate whether Ada would prevent the lung tissue from damage development over the I-R process.